For patients who haven't good knowledge of FSGS, they may be very worried
about their health state. As there is so little information about FSGS, it is
very difficult for patients who want to have a very clearly understanding of
FSGS. So I hope this article can be some of help for patients with FSGS.
There are two types of FSGS, which can be classified as follows:
Primary (idiopathic) FSGS
FSGS with hyalinosis
Progression from minimal-change disease
Progression from immunoglobulin M (IgM) nephropathy
Progression from mesangial proliferative glomerulonephritis
Superimposed on other primary glomerulonephritis conditions (eg, membranous
glomerulonephritis, immunoglobulin A [IgA nephropathy)
Variants of primary FSGS
Collapsing form
Cellular variant (endocapillary and extracapillary hypercellularity)
FSGS with mesangial hypercellularity
FSGS with glomerular tip lesions
Secondary FSGS
Drugs
Intravenous heroin
Analgesics
Viruses
Hepatitis B
HIV
Parvovirus
Hemodynamic factors - With reduced renal mass
Solitary kidney
Renal allograft
Renal dysplasia
Renal agenesis
Oligomeganephronia
Segmental hypoplasia
Vesicoureteric reflux
Hemodynamic causes - Without reduced renal mass
Massive obesity
Sickle cell nephropathy
Congenital cyanotic heart disease
Malignancies
Lymphomas
Other malignancies
Scarring - Postinflammatory in postinfectious glomerulonephritis
Miscellaneous
Hypertensive nephrosclerosis
Alport syndrome
Sarcoidosis
Radiation nephritis
The specific reasons for why we got FSGS still unknown, the treatment for
FSGS is Immunotherapy in our hospital, which have very good effects in treating
FSGS. We have treated about 1000 patients with FSGS. There are 1200 beds in our
hospital now, as a kidney hospital with 25 history, experts in our hospital have
helped more than 20000 FSGS patients in all. So if you have FSGS, please consult
our experts on line.
